#Diverticulitis #Excellacor

Introduction to Diverticulitis

Diverticulitis is a condition of inflammation. The walls of the intestines balloon out forming pouches where undigested food particles, small seeds, [e.g. strawberries, raspberries] lodge. These pouches, called diverticula, become perforated (torn), infected and inflamed.

Diverticula, due to frequent constipation and eating a low fiber diet, which is typical in the USA, contributes to the development of diverticulitis [“itis” meaning inflammation]. Low fiber diets produce maladaptive changes in the colon resulting in increased pressure that can cause pouches to form at weak points in the wall of the colon. When there is insufficient fiber to add bulk and the inability to soften stool, stools are harder to pass.

Other causes that are associated with diverticulitis include a family history of the disease, thyroid deficiency, emotional stress causing colon spasm, hidden food allergies, and may be related to vascular disorders or gallbladder disease as well as obesity, or a poor diet . It is well known that stress and smoking make symptoms worse.

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Diverticulitis is usually associated with constipation, but bouts of diarrhea should not be overlooked as well. Diarrhea is the result of insult and injury to the cells of the small intestine.

With diarrhea there is dehydration and a loss of electrolytes. Fluids are very important for colon health as fluids help rid the body of toxins. Tests should be conducted to see if there is a bacterial infection, viral infection, or parasitic infection.  

**NOTE: artificial sweeteners cause diarrhea.

Symptoms of diverticulitis include episodes of lower abdominal pain and cramping, changes in bowel habits, and a sense of fullness in the abdomen. With chronic or severe cases, there might be fever, tenderness and rigidity of the abdomen over the area of the intestine involved.

People may experience:

Pain areas: in the abdomen

Gastrointestinal: bloating, blood in stool, change in bowel habits, constipation, diarrhea, indigestion, nausea, vomiting, or flatulence

Whole body: chills, fever, or loss of appetite

Also common: cramping

Consult a doctor for medical advice

Sources: Mayo Clinic

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#Parkinson’s #disease is an irreversible #nervous #system #disorder TRY #Serracor-NK

   

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  Parkinson’s disease is an irreversible nervous system disorder that primarily affects musculo-skeletal movement. While the progressive disorder can cause stiffness and slow movements, signs and symptoms vary greatly among patients.

Common symptoms include: 
Tremors
Slowed Movement
Rigid Muscles
Impaired balance
Impaired Posture 
Speech changes

Parkinson’s disease is caused by the gradual breakdown of brain cells called neurons. The loss of neurons leads to a decrease in dopamine levels, leading to abnormal brain activity. Only 5% of Parkinson’s cases occur before the age of 40, with incidence increasing with age. A majority of cases seem to be sporadic and of unknown origin – however, like most diseases, there is a significant genetic component.

Recent theories about the causes of Parkinson’s disease are based on abnormal fibrin production and deposition in the brain. Also known as the “amyloid hypothesis,” researchers have been looking at the role of abnormal protein aggregation in Parkinson’s and other neurodegenerative disorders, like Alzheimer’s disease. There seems to be a correlation between fibrin deposits and brain tissue degeneration, suggesting that the fibrin contributes to cellular death in these diseases. Similar to Alzheimer’s disease and Huntington’s disease, Parkinson’s disease involves the mis-folding of a specific protein, eventually leading to fibrin deposits.

There are several theories as to the underlying cause of Parkinson’s, from oxidative damage to environmental toxins – but the latest research seeks to understand why the dopamine-containing neurons are affected so greatly. In fact, by the time a Parkinson’s patient presents with symptoms, about 70% of the dopamine-containing neurons have already been lost. Current therapies aim to address dopamine deficiencies, but future therapies seek to address the prevention of neuron degeneration (cellular death of the brain cells).

Although systemic enzymes are implicated for the treatment of Alzheimer’s disease, their use in similar neurodegenerative diseases has yet to be studied. Specifically nattokinase, which has been shown to directly dissolve the aggregated protein found in Alzheimer’s disease, may be an option available to those suffering from Parkinson’s disease.

 Nattokinase is a natural systemic enzyme that helps to decrease fibrin levels in the blood. Nattokinase is also able to reach areas where fibrin has already accumulated – helping to restore blood flow and inhibiting cellular death due to oxidative damage. Perhaps the most promising aspect of systemic enzyme therapy is its ability to be used concurrently with other medications. The only restriction is upon the use of nattokinase, which should be discussed with a doctor prior to adding it to a regimen with prescription blood thinners. However, it should be noted that systemic enzymes do not cause adverse side effects or drug-drug/drug-nutrient interactions.

 

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